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Study finds skin organoid blood vessels form functional microvascular networks

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Researchers at Boston Children’s Hospital and Brigham and Women’s Hospital have developed a lab-grown skin model with blood vessels that self-organize, respond to inflammation, and regenerate after injury, reducing reliance on animal testing.

Key Findings

  • Single blood vessel cells in lab-grown skin organoids can self-organize into complex microvascular networks that grow and mature over time.
  • These networks function similarly to native human skin, responding to inflammatory compounds and regenerating after injury.
  • The study, published in The American Journal of Pathology, is the first to demonstrate microvascular responses to inflammatory stimuli and injury in this system.

Background

Skin organoids are small, self-organizing multicellular systems that replicate many anatomical features and biological functions of human skin. Investigators at Boston Children's Hospital developed a procedure using stem cells to generate hair-forming human skin in a dish. Researchers from Brigham and Women's Hospital used these organoids to study skin development and disease.

Details

  • Vascular endothelial cells appear as early as six days into the organoid differentiation process and persist for months.
  • Organoids produce molecules important for initiating and maintaining small blood vessel growth.
  • As organoids grow, vessels mature and become surrounded by mural cells, similar to native skin.
  • Molecular triggers of inflammation caused organoids to express proteins for immune cell homing and release inflammatory mediators.
  • Organoid blood vessels regenerated after traumatic injuries.

Limitations and Future Applications

  • The organoid vessels showed a molecular signature of small arteries but not veins or lymphatic vessels, indicating the system is not perfect.
  • The model may be useful for studying microvasculopathy, such as in diabetes.
  • Supporting NIH and FDA initiatives to create sophisticated human disease models, this work reduces reliance on animal models.
  • The system allows study of pathways controlling skin blood vessel growth and function, with potential for therapeutic modulation in inflammatory skin conditions and chronic wounds.